NADPH Oxidase 4 Regulates Inflammation in Ischemic Heart Failure: Role of Soluble Epoxide Hydrolase

Role of Soluble Epoxide Hydrolase in Postischemic Recovery of Heart Contractile Function

Frontiers Soluble Epoxide Hydrolase Inhibition for Ocular Diseases: Vision for the Future

NADPH Oxidase 4 Induces Cardiac Fibrosis and Hypertrophy Through Activating Akt/mTOR and NFκB Signaling Pathways

NADPH Oxidase 4 Regulates Inflammation in Ischemic Heart Failure: Role of Soluble Epoxide Hydrolase

NADPH Oxidase-4 Driven Cardiac Macrophage Polarization Protects Against Myocardial Infarction–Induced Remodeling

NADPH Oxidase 4 Induces Cardiac Fibrosis and Hypertrophy Through Activating Akt/mTOR and NFκB Signaling Pathways

5-Methoxytryptophan attenuates postinfarct cardiac injury by controlling oxidative stress and immune activation - Journal of Molecular and Cellular Cardiology

Nox4 and soluble epoxide hydrolase synergistically mediate homocysteine-induced inflammation in vascular smooth muscle cells - ScienceDirect

NADPH Oxidase 4 Regulates Inflammation in Ischemic Heart Failure: Role of Soluble Epoxide Hydrolase

Frontiers Post-myocardial infarction fibrosis: Pathophysiology, examination, and intervention

Soluble Epoxide Hydrolase Regulation of Lipid Mediators Limits Pain